Front. Cell. Infect. Microbiol. Frontiers in Cellular and Infection Microbiology Front. Cell. Infect. Microbiol. 2235-2988 Frontiers Media S.A. 10.3389/fcimb.2017.00480 Microbiology Correction Corrigendum: Genome Reduction for Niche Association in Campylobacter Hepaticus, A Cause of Spotty Liver Disease in Poultry Petrovska Liljana 1 * Tang Yue 1 † Jansen van Rensburg Melissa J. 2 3 † Cawthraw Shaun 1 Nunez Javier 4 Sheppard Samuel K. 5 Ellis Richard J. 1 Whatmore Adrian M. 1 Crawshaw Tim R. 1 Irvine Richard M. 1 1Bacteriology, Animal and Plant Health Agency Weybridge, Addlestone, United Kingdom 2Department of Zoology, University of Oxford, Oxford, United Kingdom 3NIHR Health Protection Research Unit in Gastrointestinal Infections, University of Oxford, Oxford, United Kingdom 4Veterinary Surveillance, Animal and Plant Health Agency Weybridge, Addlestone, United Kingdom 5Department of Biology and Biotechnology, The Milner Centre for Evolution, University of Bath, Bath, United Kingdom

Edited and reviewed by: Alain Stintzi, University of Ottawa, Canada

*Correspondence: Liljana Petrovska liljana.petrovska@apha.gsi.gov.uk

†These authors have contributed equally to this work.

 14 11 2017 2017 7 480 21 08 2017 03 11 2017 Copyright © 2017 Petrovska, Tang, Jansen van Rensburg, Cawthraw, Nunez, Sheppard, Ellis, Whatmore, Crawshaw and Irvine. 2017 Petrovska, Tang, Jansen van Rensburg, Cawthraw, Nunez, Sheppard, Ellis, Whatmore, Crawshaw and Irvine

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 A corrigendum on Genome Reduction for Niche Association in Campylobacter Hepaticus, A Cause of Spotty Liver Disease in Poultry by Petrovska, L., Tang, Y., Jansen van Rensburg, M. J., Cawthraw, S., Nunez, J., Sheppard, S. K., et al. (2017). Front. Cell. Infect. Microbiol. 7:354. doi: 10.3389/fcimb.2017.00354 Campylobacter hepaticus spotty liver disease poultry genome reduction niche adaptation Department for Environment, Food and Rural Affairs10.13039/501100000277

In the original article, there was a mistake in Table 3 as published. Table 3 had additional genes inserted for isolates S11-0036, S11-0038, S11-0069, and S12-0071. Isolate S12-002 should not be included in Table 3.

Presence of pathogenicity-related genes in C. hepaticus.

Protein (name) Protein ID S11-0036 S11-0038 S11-0069 S11-0071 S10-0209 S12-1018 S11-5013 S11-010 S12-0322
F2 F2 F4 F4 F1 F1 F1 F1 F5
MCP EAQ73158
TrkA ABS44147
CHP1 EAQ72353
CHP2 EAQ72298
HP1 EAQ71971
HAD-superfamily phosphatase, subfamily IIIC EAQ72583
Putative 3-oxoacyl- synthase ABS43995
Methyltransferase CAL35414
DNA adenine methylase AAW34814
HP2 EAQ72552
HP3 HP3
Putative DNA-binding protein AAW34848
Putative acyl carrier protein CAL35413
Putative acyl carrier protein AAW35934
CHP3 EAQ71755
Putative SAM domain containing methyltransferase CAL35414
CHP4 EAQ72353
cpp14 AAR29498.1
cpp17 AAR29501.
cpp22 AAR29505.
cpp18 AAR29502.
cpp47 AAR29530.
cpp45 AAR29528.
cpp29 AAR29512.
cpp13 AAR29497.1
pTet AY714214.
cmgB3/4 AAR29514.1

Purple, present; blank, absent; orange, plasmid pTet (pCC31, AY394560.1) related proteins, dark blue, proteins not present in C. jejuni 11168. Farms 1, 2, 4, and 5 are indicated (F1, F2, F4, and F5).

Additionally, there was an incorrect sentence. Incorrect sentence describing the number of RNA coding sequences and the GC content. A correction has been made to Results, C. hepaticus Isolates Have Reduced Genomes, Paragraph Number One and appears below.

The C. hepaticus isolates had a lower number (average of 44) of RNA coding sequences and a lower GC content (average of 28.4%) in comparison to the C. jejuni reference genomes (average of 52.4 and 30.5%, respectively).

Similarly, there was an incorrect sentence. Incorrect sentence describing the genes related to pathogenicity of C. hepaticus. A correction has been made to Results, genes related to the Pathogenicity of C. hepaticus, Paragraph Number One and appears below.

The UK C. hepaticus isolates contained relatively few genes linked to pathogenesis: 5 were identified in the genomes of S11-0036, S11-0069, S11-0071 and (from farms 2, and 4); 6 in S11-0038 (farm 2); 15 in S10-0209, S12-1018, S11-5013, and S11-010, (farm 1); and 7 in isolate S12-0322 (farm 5; Table 3). The cpp and cmgB3/4 genes, both components of the pTet plasmid (Batchelor et al., 2004), and a complete pTet plasmid (Batchelor et al., 2004) sequences were identified in isolates S11-010, and S12-0322 (Table 3).

Finally, in incorrect spelling of metabolism was used, we omitted “the” and misspelled “rich.” A correction has been made to Discussion, Paragraph Number Four and appears below.

Furthermore, Stahl and co-workers found that the ability to metabolize L-fucose in vivo provided C. jejuni with competitive advantage during colonization of the piglet infection model. Similar was not observed in the chick commensal model (Stahl et al., 2011), suggesting potential niche specific advantage for colonization in the L-fucose rich environment in the pig small intestine and cecum.

The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way.

Conflict of interest statement

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.