Front. Cardiovasc. Med.Frontiers in Cardiovascular MedicineFront. Cardiovasc. Med.2297-055XFrontiers Media S.A.10.3389/fcvm.2021.793246Cardiovascular MedicineCorrectionCorrigendum: Extracorporeal Cardiac Shock Waves Therapy Improves the Function of Endothelial Progenitor Cells After Hypoxia Injury via Activating PI3K/Akt/eNOS Signal PathwayWangMingqiang1†YangDan1†HuZhao1ShiYunke1MaYiming1CaoXingyu1GuoTao2CaiHongbo3*CaiHongyan1*1Department of Cardiology, The First Affiliated Hospital of Kunming Medical University, Kunming, China2Department of Cardiology, Yunnan Fuwai Cardiovascular Hospital, Kunming, China3Department of Vascular Surgery, The First Affiliated Hospital of Kunming Medical University, Kunming, China
Edited by: Xiaofeng Yang, Temple University, United States
Reviewed by: Zhao-Jun Liu, University of Miami, United States; Feng Dong, Northeast Ohio Medical University, United States
*Correspondence: Hongbo Cai hongbocai@hotmail.comHongyan Cai hyflykm@sina.com
This article was submitted to Cardiovascular Therapeutics, a section of the journal Frontiers in Cardiovascular Medicine
†These authors have contributed equally to this work and share first authorship
This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
A Corrigendum on Extracorporeal Cardiac Shock Waves Therapy Improves the Function of Endothelial Progenitor Cells After Hypoxia Injury via Activating PI3K/Akt/eNOS Signal Pathway by Wang, M., Yang, D., Hu, Z., Shi, Y., Ma, Y., Cao, X., Guo, T., Cai, H., and Cai, H. (2021). Front. Cardiovasc. Med. 8:747497. doi: 10.3389/fcvm.2021.747497extracorporeal cardiac shock wavesendothelial progenitor cellscell functionPI3K/Akt/eNOS signaling pathwayshypoxia injurynitric oxide (NO)
In the original article, there was a mistake in Figure 4 as published. Because of our carelessness in combining images, we put the wrong images on Figures 4C and E. The corrected Figure 4 appears below.
ECSW improve the adhesive, migratory, and tube formation capacity of EPCs after hypoxic injury by activating PI3K/Akt/eNOS signaling pathway. (A,C,E) Representative images of EPCs adhesive, migratory, and tube formation in each group under a microscope. (A,E) Scale bar = 100 μm; (C) Scale bar = 200 μm. (B,D,F) Quantitative analysis of the adhesive, migratory, and tube formation of EPCs in each group. Data are presented as mean ± SD, N = 3. *P < 0.05 vs. group control, **P < 0.05 vs. group hypoxia, #P < 0.05 vs. group hypoxia + ECSW.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Publisher's Note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.